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Because of the potential role of free radicals in ALS and the upset in function of the superoxide dismutase enzyme in the familial form of ALS, previous studies have suggested that an increase in free radicals may impair the function of GLT-1 [77]
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Immunoglobulin subclasses 1 and 4 of the DSA are associated with enhanced C1 binding capacity and the degree of complement activation and may therefore determine the severity of the injury
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