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However, it remains controversial whether fibrosis and inflammation are the primary pathological drivers of keloid formation or secondary phenomena triggered by upstream events, such as genetic susceptibility, dysregulation of the TGF-β signaling pathway, or abnormal fibroblast proliferation (Zhang et al., 2009)

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Potential therapies Although some researchers have attempted to establish a treatment for ME/CFS and no reproducible results have been found (60), some treatments seem promising in ME/CFS ( Figure 13 )